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It is important to remember that there is a wide range of normal variability in the 12 lead ECG. The following "normal" ECG characteristics, therefore, are not absolute. It takes considerable ECG reading experience to discover all the normal variants. Only by following a structured "Met hod of ECG Interpretation" (Lesson II) and correlating the various ECG findings with the particular patient's clinical status will the ECG become a valuable clinical tool.
Topics for Study:
How to calculate the heart rate on ECG paper
Because ECG paper moves at a standardized 25mm/sec, the vertical lines can be used to measure time. There is a 0.20 sec between 2 of the large lines. Therefore, if you count the number of heart beats (QRS complexes) in between 30 large boxes (6 seconds) and multiply by 10, you have beats per minute. Conveniently, ECG paper usually has special markings every 3 seconds so you don't have to count 30 large boxes.
There is, however, an easier and quicker way to estimate the heart rate. As seen in the diagram below, when QRS complexes are 1 box apart the rate is 300 bpm. 2 boxes apart...150 bpm, etc. So if you memorize these simple numbers you can estimate the heart rate at a glance!
Bazett's Formula: QTc = (QT)/SqRoot RR (in seconds)
Poor Man's Guide to upper limits of QT: For HR = 70 bpm, QT<0.40 sec; for every 10 bpm increase above 70 subtract 0.02 sec, and for every 10 bpm decrease below 70 add 0.02 sec. For example:QT < 0.38 @ 80 bpm
QT < 0.42 @ 60 bpm
(Normal ECG is shown below - Compare its waveforms to the descriptions below)
Click to view
P duration < 0.12 sec
P amplitude < 2.5 mm
Frontal plane P wave axis: 0o to +75o
May see notched P waves in frontal plane
QRS Complex
The QRS represents the
simultaneous activation of the right and left ventricles, although most
of the QRS waveform is derived from the larger left ventricular musculature.
QRS duration < 0.10 sec
QRS amplitude is quite variable from lead to lead and from
person to person. Two determinates of QRS voltages are:
Size of the ventricular chambers (i.e., the larger the chamber, the larger the voltage)
Proximity of chest electrodes to ventricular chamber (the closer, the larger the voltage)
The normal QRS axis range (+90 o to -30 o ); this implies that the QRS be mostly positive (upright) in leads II and I.
Normal q-waves reflect normal septal activation (beginning on the LV septum); they are narrow (<0.04s duration) and small (<25% the amplitude of the R wave). They are often seen in leads I and aVL when the QRS axis is to the left of +60o, and in leads II, III, aVF when the QRS axis is to the right of +60o. Septal q waves should not be confused with the pathologic Q waves of myocardial infarction.
Small r-waves begin in V1 or V2 and progress in size to V5. The R-V6 is usually smaller than R-V5.
In reverse, the s-waves begin in V6 or V5 and progress in size to V2. S-V1 is usually smaller than S-V2.
The usual transition from S>R in the right precordial leads to R>S in the left precordial leads is V3 or V4.
Small "septal" q-waves may be seen in leads V5 and V6.
Normal ST segment elevation: this occurs in leads with large S waves (e.g., V1-3), and the normal configuration is concave upward. ST segment elevation with concave upward appearance may also be seen in other leads; this is often called early repolarization, although it's a term with little physiologic meaning (see example of "early repolarization" in leads V4-6):
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Convex or straight upward ST segment elevation (e.g., leads II, III, aVF) is abnormal and suggests transmural injury or infarction:
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ST segment depression is always an abnormal finding, although often nonspecific (see ECG below):
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ST segment depression is often characterized as "upsloping", "horizontal", or "downsloping".
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The normal U Wave: (the most neglected of the ECG waveforms)
U wave amplitude is usually < 1/3 T wave amplitude in same
lead
U wave direction is the same as T wave direction in that lead
U waves are more prominent at slow heart rates and usually best
seen in the right precordial leads.
Origin of the U wave is thought to be related to
afterdepolarizations which interrupt or follow repolarization.